More bird flu stuff...and I' really getting tired of having dreams like this.
Brian, your drawing says "baby dies, 105.2 degrees" A baby has just died from H5N1, and it was reported that her boby temperature was 105.2 degrees, also you wrote "DjiBout" in the upper right corner of your dream! Please see the attached news story.
Charles J.
Hi, thanks for this link, I will post your information...For the past several dream I have had about bird flu this week, I'm amost certian that the 'human to human' mutation has happened.
Bird Flu Strikes Indonesian Family Again
The Associated Press
Sunday, May 14, 2006; 1:50 PM
JAKARTA, Indonesia -- A fifth member of an Indonesian family has died of bird flu, according to local tests, a senior medical official said Sunday.
The other four died from the disease early last week, said Nyoman Kandun, head of the Health Ministry's office of communicable disease control. In all, eight members of the family in Tanah Karo village on Sumatra are suspected of having contracted the virulent H5N1 bird flu virus.
Indonesia's official human death toll from the virus is now 25, the world's second-highest after Vietnam, which recorded about four dozen deaths but where international health experts said earlier this year that outbreaks of H5N1 infections in people and poultry had been largely stamped out.
Officials pay particular attention to cases of multiple related deaths such as those in Indonesia. The virus now is almost always transmitted from birds to humans, and experts study "cluster" cases looking for signs that H5N1 may have mutated into a form easily passed between humans _ a scenario that many fear could trigger a global human pandemic.
Kandun said samples from the patients had been sent to a World Health Organization-accredited lab in Hong Kong for confirmation that they died of bird flu.
He also revealed that a sixth family member died previously, but samples from the body had not been taken for laboratory investigation. "The person died earlier and has been buried," he said.
Asked whether doctors would obtain samples from the body, Kandun declined to comment.
"We are continuing to investigate this case," he said. "We are trying to find the source of the virus."
He said investigators were checking poultry near the family's village, since veterinarians had found no signs of bird flu among chickens and other animals in the village itself.
International experts hope Vietnam's campaign to vaccinate its poultry against H5N1 will serve as an example for other countries in dealing with the virus, which has killed more than 100 people in all.
Once the epicenter for bird flu, Vietnam hasn't had seen any people infected since November and there haven't been any poultry outbreaks since December.
"We are actually disease-free in Vietnam for the moment," Hans Troedsson, the World Health Organization representative in Vietnam, said in late March. "We're probably not virus-free, but what the mass vaccination has done is reduce the virus load in the environment _ we have less virus circulating."
Vietnam started its nationwide eradication campaign in August 2005. Officials in the poor communist nation say they vaccinated millions of chickens and ducks, slaughtered millions more and educated citizens about the disease.
Outside experts, however, caution that immunization is not a simple solution.
It is so expensive that "you just can't keep it up," said Peter Roeder, an animal health expert with the U.N. Food and Agriculture Organization who advises Asian countries on bird flu.
5.13.2006
More info on yesterdays prediction...
H1N1 is The 1918 virus...
For some reason I cannot cut and paste from this link...worth a read though, I think!
Love and Health,
Mariella
reply
Hi, thanks will post this link.
Brian
H1N1-influenza as Lazarus: Genomic resurrection from the tomb of an unknown
Joshua Lederberg*The Rockefeller University, 1230 York Avenue, New York, NY 10021-6399
The 1918-1919 pandemic of H1N1 virus influenza was the greatest acute plague of the 20th century. Incurring over 20 millionhuman fatalities, however, was not a good strategy for sustainingthe evolutionary fitness of the virus, because it is no longerextant; whereas, say, measles and chickenpox remain with us withno evidence of remarkable genetic change, although this may becomemore evident if they were to face total or near eradication throughvaccination programs. The folly of flu virulence remains our chagrin,because the threat always looms over us that this family of viruses,endemic in birds, again may generate human-lethal gene reassortments.We had valid scares about that contingency with the appearanceof H5N1 variant flu in Hong Kong just 3 years ago. Influenza canbe regarded as a zoonosis prevalent in birds, many of them worldtravelers, with occasional outbreaks in humans and other animalsmainly rooted in nature's own experiments in genetic engineering.Special importance is attached to reassortments between bird-and human-adapted strains most likely to occur in habitats withclose contact between birds, e.g., ducks, humans, and swine (asa mixing reservoir; ref. 1). For these reasons, high urgencyattaches to efforts to resurrect genetic information about thesingularities of H1N1-1918. The intact virus is nowhere to befound, but genomic fragments can still be detected sensitivelyand diagnosed. Exemplifying the latest technical advances in theuse of DNA amplification, reverse-transcriptase-PCR (RT-PCR),Jeffery Taubenberger and his associates at the Armed Forces Instituteof Pathology initiated the tour de force of recovering sequencesof flu from paraffin-embedded pathological specimens preservedsince 1918 in the AFIP collections (2). These sources thenwere augmented by samples from frozen remains of an Inuit womanwho succumbed to the flu in 1918 and was buried in permafrostat Brevig Mission on the Seward Peninsula of Alaska's westerncoast, not far from the Bering Strait. This nameless woman hasleft an indelible mark on world medical history (3). Now, asreported in this issue, the AFIP team has joined forces with teamsfrom the U.S. Department of Agriculture and the Peter Palese/AdolfoGarcía-Sastre groups at Mt. Sinai Medical School in a furtherquest for the RNA sequences of H1N1-1918 that might account forits historic human virulence (4).
The flu genome comprises about 13,500 bases of single-stranded RNA, disposed in eight segments varying from approximately900 to 2,341 each. This genome is only a few millionths of thecomplexity of the human genome, but it is organized with greatefficiency, lacks "junk R/DNA," and encodes for a short dozenof identified gene products (Fig. 1). Many strains of flu havebeen sequenced fully; this feat will be achieved for H1N1-1918with arduous labor, because the RNA, although frozen, is fragmentedinto snippets no larger than approximately 120 bases each. Thepractical way now available is to devise probes by using segmentsfrom extant flu strains, guessing at possible homologous strings,or synthesizing probes with calculated degeneracy. Until a completegenomic sequence is achieved, and it is hard to see how that willbe authenticated, it is possible even that H1N1-1918 containsextraneous inserted sequences quite foreign to the canonical flustrains. Very reasonably, initial efforts focus on flu genes alreadyidentified in viruses recovered from recent outbreaks in humans,birds, swine, and other animals.
Previous work has focused on two well studied gene products: hemagglutinin (HA) and neuraminidase (NA), which dominate thesurface specificities of the virus and underlie most of its taxonomy(e.g., H1N1 refers to type 1 hemagglutinin, type 1 neuraminidase).These gene products are also the chief determinants of specificityin vaccine prophylaxis for flu strains circulating at any giventime. HA variation can account for fluctuations of virulence andhost specificity of extant flu viruses. However, nothing remarkablewas seen in the HA or the NA of H1N1-1918. The next gene to bescrutinized now is NS1 (nonstructural protein 1), which the Palese/García-Sastregroups have fingered recently as an interferon antagonist andas gene essential for flu virulence in a mouse model. A reasonableconjecture was that the hypervirulence of H1N1-1918 might be lodgedin its NS1, and this might be revealed in reinsertions of the1918-NS1 segment into mouse-adapted flu strains. This challengingconstruct was generated in the laboratory
one hastens to footnote,under BL-3+ conditions, and under the USDA's stern regulatoryscrutinyand tested in mice. The unexpected and perhaps disappointingresult was the mitigation not enhancement of virulence in thisspecies. The incapacitation of the NS1-virulence function in themouse was ascribed to interaction with its host factors; the othervariable would be other elements of the genome of the mouse-adaptedflu strain. NS1 singularity for the human virulence of H1N1-1918is neither falsified nor corroborated by thesefindings.
There still remain a handful of gene candidates, including the polymerases essential for the replication of the virus. Thislabel does not preclude any of them from also functioning in networksand pathways that are expressed as virulence. It should cautionus about the nominalist fallacy to recall that the 
crystallinof the bird's lens does double duty as argininosuccinate lyase,an enzyme in the ureacycle.
In principle, the NS1 hypothesis (and its alternatives) might be tested by using similar gene constructs based on flu virusesadapted to other animal species, including primates, and challengingthe corresponding hosts. Negative results would be as inconclusiveas those with the mouse. Positive results, namely the associationof hypervirulence with a gene sequence borrowed from N1H1-1918,would be a great advance in medical science and would offer constructivemodels for the development of prophylactic and therapeutic measures.They would also induce great alarm about the potential hazardsto human health, if humans were also susceptible, and the virusmight escape. Any such experiments should be done with strainsfor which current vaccines are disseminated widely and have proveneffectiveness.
To conduct such experiments with human-adapted strains and challenge to human subjects as the probative step, is well nighunthinkable. But nature is under no such restraint! The currentresults are a caution to look closely at the involvement of NS1(as well as HA and NA) variation in natural outbreaks in manyspecies and to look out for their reassortment into human strains.In addition, it might be well to undertake a special search forclose homologues to 1918-NS1 in viruses circulating in avian andother species, in which they may appear to be benign in theircurrent hosts (as in the present mouse experiments). That wouldbe nature's inverse of the currentreport.
The publication by Basler et al. (4) will attract great admiration for its technical finesse and will serve as an exampleof the fruits from convergence of natural history, field exploration,clinical insight, and sophisticated molecular wizardry. It alsowill awaken anxieties about the obvious opportunities for abuse.The really fateful step was taken with the very first cultivationof pathogenic bacteria and viruses a century agoperhaps mostimportantly with the discovery of the concepts of germs and communicablediseases. The notion of using ever more sophisticated technologyfor intentionally constructing or reconstructing ever more pathogenicvariants lends further weight to that anxiety. The great debateof the mid-1970s led to sensible measures for the regulation ofrecombinant DNA research. There has been increasing understandingthat some of nature's pathogens deserve equal or greater respect.We should be sure that we continue to devote as much reasonedingenuity to the design of safeguards and to informed and transparentthird-party scrutiny of potential hazards as we do generally tothe authentication of scientific claims. We cannot afford to foregothe deepest research into the plagues that beset humankind. Norcan we afford to blunder into mistakes that will do primary injuryto bystanders and incur incommensurate socialsanctions.
My deepest anxieties pertain to the smoldering technology and arms race that attends the power struggles in the Middle Eastand the economic instabilities of the former Soviet Union. Althoughthe 1975 Biological Weapons Convention (BWC) has demilitarizedthe main drivers of bioweaponry technical advance, in the U.S.and in the overt activities of other formidable powers, the BWChas not been enforced successfully against Iraq and is more orless openly flouted in a handful of other countries. The UnitedNations (UN) Security Council is too splintered on other issuesto take a firm stand on the defiance by Iraq of the UN-mandatedinspections. It would not be child's play for defiant small countriesto adopt advanced biotechnology into their weapons programs. Butwe have seen that the climactic high-science successes in onedecade become fodder for high-school projects in the next. Influenzais an unlikely candidate for rational weapons development, becausenew strains promptly embrace the world. But that logic is insufficientreason to neglect the contingency. More likely similar principleswould be applied to more governable bioagents, but any bioagentsin warfare are an affront and a threat to the entire human species.Informed professionals throughout the world should be leadingcampaigns to insist on universal compliance with the BWC as amajor bulwark of human health and associating that with the mostpositive measures to apply advanced biotechnology in a constructiveway for dealing with nature's continuedscourges.
5.13.2006
Morning Brian!
Just to let you know there has been a very recent news report about just this vaccine...
reply
Thanks, will post this link.
Brian
Bird Flu Vaccine Looks Safe, Effective
But it might not work against new strains of the virus, experts warnBy Amanda Gardner
HealthDay Reporter
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